Joseph L. Witztum - Concepedia

Author

Joseph L. Witztum

University of California San Diego ORCID

Also Known As

J L Witztum, J Witztum, J. L. Witztum, J. Witztum, J.L Witztum, J.L. Witztum, JOSEPH L. WITZTUM, Jian Wang, Joseph L Witztum, Joseph L. WITZTUM

603

Publications

92.4K

Citations

157

H-Index

230

Concepts

All Affiliations

University of California San Diego Ionis Pharmaceuticals (United States)

About

Joseph L. Witztum is an author at University of California San Diego specializing in medicine, atherosclerosis, and oxidative stress.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	94	174	36.2K
2	Health Sciences	30	35	7.6K
3	Public Health	24	26	3.2K
4	Natural Sciences	16	16	2.1K
5	Engineering	4	4	648

Joseph L. Witztum

369

Publications

65.9K

Citations

139

H-Index

Page 1

	Year	Citations
Evidence for the presence of oxidatively modified low density lipoprotein in atherosclerotic lesions of rabbit and man. Seppo Ylä‐Herttuala, Wulf Palinski, Michael E. Rosenfeld, Journal of Clinical Investigation Vascular DiseasePathologyHyperlipidemiaLow DensityOxidative Stress	1989	1.9K
Modification of low density lipoprotein by endothelial cells involves lipid peroxidation and degradation of low density lipoprotein phospholipids. Urs P. Steinbrecher, Srinivasan Parthasarathy, David S. Leake, Proceedings of the National Academy of Sciences Endothelial CellsLipid PeroxidationCu2+ ConcentrationRedox BiologyOxidative Stress	1984	1.6K
Fatty streak formation occurs in human fetal aortas and is greatly enhanced by maternal hypercholesterolemia. Intimal accumulation of low density lipoprotein and its oxidation precede monocyte recruitment into early atherosclerotic lesions. Claudio Napoli, Francesco Paolo D’Armiento, Francesco Paolo Mancini, Journal of Clinical Investigation HyperlipidemiaOxidative StressMetabolic SyndromeHuman Fetal AortasAtherosclerosis	1997	986
Lipoprotein(a) Reduction in Persons with Cardiovascular Disease Sotirios Tsimikas, Ewa Karwatowska‐Prokopczuk, Ioanna Gouni‐Berthold, New England Journal of Medicine	2020	924
Expression of monocyte chemoattractant protein 1 in macrophage-rich areas of human and rabbit atherosclerotic lesions. Seppo Ylä‐Herttuala, Beth Lipton, Michael E. Rosenfeld, Proceedings of the National Academy of Sciences Vascular DiseaseImmunologyPathologyCellular PhysiologyInflammation	1991	877
Interleukin-4-dependent production of PPAR-γ ligands in macrophages by 12/15-lipoxygenase Jannet T. Huang, John S. Welch, Mercedes Ricote, Nature InflammationCytokineMedicineImmunologyPpar-γ Ligands	1999	842
Oxidized Phospholipids, Lp(a) Lipoprotein, and Coronary Artery Disease Sotirios Tsimikas, Emmanouil S. Brilakis, Elizabeth R. Miller, New England Journal of Medicine	2005	753
Structural Identification by Mass Spectrometry of Oxidized Phospholipids in Minimally Oxidized Low Density Lipoprotein That Induce Monocyte/Endothelial Interactions and Evidence for Their Presence in Vivo Andrew D. Watson, Norbert Leitinger, Mohamad Navab, Journal of Biological Chemistry	1997	752
Pneumococcal vaccination decreases atherosclerotic lesion formation: molecular mimicry between Streptococcus pneumoniae and oxidized LDL Christoph J. Binder, Sohvi Hörkkö, Asheesh K. Dewan, Nature Medicine VaccinationInflammationStreptococcus PneumoniaePneumococcal VaccinationPathogenesis	2003	730
Antisense oligonucleotides targeting apolipoprotein(a) in people with raised lipoprotein(a): two randomised, double-blind, placebo-controlled, dose-ranging trials Nicholas J. Viney, Julian C. van Capelleveen, Richard S. Geary, The Lancet Dose-ranging TrialsPharmacologyHyperlipidemiaPharmacotherapyDyslipidemia	2016	730

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