Masataka Mori - Concepedia

Author

Masataka Mori

Kumamoto University ORCID

Also Known As

M MORI, M Mori, M. MORI, M. Mori, M., Mori, MASATAKA MORI, Masataka MORI, Masataka Mori, Masatomo Mori, Miyuki Mori

573

Publications

22.5K

Citations

H-Index

326

Concepts

All Affiliations

Kumamoto University Chiba University Gunma University Japan Tobacco (Japan)

About

Masataka Mori is an author at Kumamoto University specializing in medicine, biochemistry, and natural sciences.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	45	126	7.6K
2	Natural Sciences	35	69	3.4K
3	Engineering	15	22	524
4	Health Sciences	14	18	957
5	Language Studies	1	1	12

Masataka Mori

270

Publications

16.8K

Citations

H-Index

Page 1

	Year	Citations
Targeted disruption of the Chop gene delays endoplasmic reticulum stress–mediated diabetes Seiichi Oyadomari, Akio Koizumi, Tomomi Gotoh, Journal of Clinical Investigation	2002	865
Targeted disruption of the Chop gene delays endoplasmic reticulum stress–mediated diabetes Seiichi Oyadomari, Akio Koizumi, Tomomi Gotoh, Journal of Clinical Investigation ApoptosisCell DeathInsulin SignalingOxidative StressEr Overload	2002	743
Nitric oxide-induced apoptosis in pancreatic β cells is mediated by the endoplasmic reticulum stress pathway Seiichi Oyadomari, Masaki Takiguchi, Tomomi Gotoh, Proceedings of the National Academy of Sciences	2001	589
Estrogen increases in vivo leptin production in rats and human subjects Hiroyuki Shimizu, Yoshiharu Shimomura, Yoshihito Nakanishi, Journal of Endocrinology FertilityGynecologyLeptin ProductionFemale Reproductive FunctionMenstrual Cycle	1997	476
Replacement of lysine residue 1030 in the putative ATP-binding region of the insulin receptor abolishes insulin- and antibody-stimulated glucose uptake and receptor kinase activity. Yasuhiko Ebina, Eiichi Araki, M. Taira, Proceedings of the National Academy of Sciences Insulin ReceptorReceptor Kinase ActivityInsulin SignalingLysine Residue 1030Molecular Pharmacology	1987	397
hsp70-DnaJ chaperone pair prevents nitric oxide- and CHOP-induced apoptosis by inhibiting translocation of Bax to mitochondria Tomomi Gotoh, Kazutoyo Terada, Seiichi Oyadomari, Cell Death and Differentiation BiochemistryMitochondrial FunctionNatural SciencesChop-induced ApoptosisApoptosis	2004	379
Peripheral Administration of Nesfatin-1 Reduces Food Intake in Mice: The Leptin-Independent Mechanism Hiroyuki Shimizu, Sinsuke Oh-I, Ko Hashimoto, Endocrinology	2009	311
Nationwide survey of HTLV‐I–associated myelopathy in Japan: Association with blood transfusion Mitsuhiro Osame, Robert S. Janssen, Hiroaki Kubota, Annals of Neurology	1990	265
Endoplasmic reticulum stress response is involved in nonsteroidal anti-inflammatory drug-induced apoptosis Shinji Tsutsumi, Tomomi Gotoh, Wataru Tomisato, Cell Death and Differentiation InflammationReductive StressAnti-inflammatoryAutoimmune DiseaseMedicine	2004	250
Nitric Oxide-induced Apoptosis in RAW 264.7 Macrophages Is Mediated by Endoplasmic Reticulum Stress Pathway Involving ATF6 and CHOP Tomomi Gotoh, Seiichi Oyadomari, Kazutoshi Mori, Journal of Biological Chemistry	2002	238

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